What Is Male Hypogonadism?

Reading Time: 19 minutes

 

Male Hypogonadism

 

What is Hypogonadism? (Gonadal deficiency)

From the Conscious Evolution Institute

Hypogonadism is any form of hormonal deficiency that results in diminished production of any of the sexual hormones. In men, Hypogonadism is the result of underperforming or non-functional testes. In women, the disorder results from ovaries that are not producing proper estrogen levels.

Not all cases are directly the result of inferior sex organs, and endocrine issues throughout the body can also lead to Hypogonadism. Hypogonadism is caused by any disruption which occurs along the hypothalamic-pituitary-gonadal axis.

This axis represents a group of organs intimately related to the production of the sex hormones (as well as a number of other essential hormones).

The testes and ovaries are the factories that produce our primary sexual hormones. The pituitary gland can send messages from the brain to the sex organs to spur or inhibit the production of sex hormones.

The hypothalamus is the master command of the system. It takes in various inputs from throughout the body and the rest of the brain and decides that Testosterone and Estrogen production should begin. The hypothalamus then sends messages to the pituitary to alert the sex organs to adjust their production rate.

Risk factors, incidence, and causes of Hypogonadism

 

 

 

Diseases related to hypogonadism belong to two distinct categories. The first category is primary hypogonadism. In this form of hypogonadism, the testes and ovaries are the sources of the disorder, and they are not producing or capable of producing healthy hormone levels. In many cases, primary hypogonadism does not affect testosterone levels, even if it results in reduced or eliminated sperm production.

Sperm are manufactured in cells known as seminiferous tubules, which are more sensitive to the damage that results from most testicular diseases. Testosterone is produced by structures known as Leydig cells, which are more insulated from potential damage.

Below is a list of common causes of primary hypogonadism:

Surgery: Sometimes, as is the case in issues such as ovarian and testicular cancer, it is necessary to remove the sex organs through either oophorectomy or testicle removal. In these patients, hormone production will cease completely, and Hormone Replacement Therapy will be necessary to maintain normal Hormone function.

Radiation: Another issue that most commonly results from cancer therapy is that radiation treatments render the testicles and ovaries under-functional or non-functional. Radiation therapy works in cancer patients by irradiating cancer cells to kill them off. The problem with radiation therapy is that it also targets healthy cells.

Radiation therapy is targeted as efficiently as possible to minimize damage, but in most cases, at least a certain level of damage to healthy tissues is unavoidable. For example, treatment of prostate cancer through radiation can damage the prostate, reducing its ability to produce healthy testosterone levels.

A research study by researchers at Mercy Medical Center in Redding, California, found that prostate cancer patients treated with prostate-bed radiation therapy had total testosterone levels, which averaged 27% less than their cohorts who underwent alternative forms of therapy.

Injury and Infection: Damage to the testicles and ovaries due to injury and infection can also result in hypogonadism.

A testicular injury such as blunt force trauma can lead directly to organ breakdown, or issues such as swelling or testicular torsion can prevent the testicles from getting proper circulation, thus killing them off or damaging them. Infections can also lead to hypogonadism by slowly damaging essential organs and blood pathways.

Infection can similarly damage the body as radiation, targeting healthy cells and breaking them down. If the infection is not treated promptly and efficiently, damage to the testicles or ovaries can be irreversible.

Kidney disease: Chronic Kidney Disease is another common cause of Hypogonadism. Hypogonadism due to this disease has been associated with decreased bone strength and osteoporosis, in addition to sexual dysfunction and depression. Studies have shown that over half of patients who suffer from kidney disease have below-normal levels of endogenous testosterone production.

At this point in scientific study, it is not yet clear exactly how chronic renal failure leads to low testosterone levels, but it upsets the fragile hypothalamic-pituitary-gonadal axis.

In addition to hypogonadism that occurs directly as a result of the disease, many medicines used to treat the disease hinder the production of Testosterone. Testosterone Hormone Replacement Therapy can reverse the effects of testosterone deficiency resulting from renal failure.

Liver disease and hypogonadism: Liver disease hurts Testosterone levels as well. It is well known in the academic community that long-term abuse of alcohol can cause the body to reduce its ability to produce Testosterone. In a small subset of cases, alcohol abuse can also lead to the conversion of Testosterone into Estrogen.

Patients who suffer from liver disease often produce lower levels of Luteinizing hormone. Luteinizing hormone is a precursor to Testosterone which leads to its production. Patients with demonstrably low levels of luteinizing hormone often need some form of Testosterone Replacement Therapy to preserve healthy function.

Causes of Primary Hypogonadism

There are a number of developmental and genetic disorders which lead to primary hypogonadism:

Klinefelter's Syndrome and Hypogonadism

In males, the most common form of genetic primary hypogonadism is the result of a disorder known as Klinefelter's syndrome. Klinefelter's syndrome results from a genetic abnormality in which the male genetic code has an additional X, resulting in an XXY configuration.

Men who suffer from Klinefelter's syndrome produce significantly less testosterone than their regular healthy male counterparts. This leads to a number of symptoms of hypogonadism, such as brittle bones, less body hair across the body, gynecomastia, and other feminized male features. Fertility is also significantly or entirely affected as well.

Testosterone Replacement Therapy is highly beneficial for children and adults with Klinefelter's syndrome because, with a mixture of hormone replacement therapy and active early intervention, these children can live happy, independent, and everyday lives.

Turner Syndrome and Hypogonadism

Turner Syndrome is among the most common genetic disorders which result in hypogonadism in girls and women. Turner syndrome is a genetic abnormality that results in either partial or total absence of the second X chromosome. Turner syndrome leads to a number of significant adverse effects, such as idiopathic short stature, underdeveloped ovaries and breasts, learning disability, and a number of other physical abnormalities dependent upon how much of the second X chromosome is still present and helpful.

Although many of the symptoms of Turner Syndrome result from Human Growth Hormone Deficiency (such as idiopathic short stature), many symptoms primarily result from estrogen deficiency. Estrogen Replacement Therapy can be used to encourage the average feminine growth of the breasts and strengthen bones that are weakened without the presence of healthy levels of estrogen.

Although Turner syndrome patients are infertile, Estrogen Replacement Therapy can also be used in conjunction with In Vitro Fertilization for a Turner patient to bear a child using donated egg cells.

Although it is clear even before birth that these patients who suffer from genetic disorders will need some level of Hormone Replacement Therapy, their syndromes produce tangible representations of the power that Estrogen and Testosterone have on healthy function how they play a significant role in making us who we are.

Auto-Immune Deficiency

Auto-Immune Deficiency has been linked to low Testosterone in males and low Estrogen in females. For this reason, Hormone Replacement Therapy can be beneficial in preventing or alleviating the symptoms of a number of Auto-Immune Disorders.

This deficiency is highly documented in patients suffering from the AIDS virus. Evidence shows that Estrogen Replacement Therapy can help prevent brain damage that can result from the progression of the AIDS virus. This can prevent dementia that often develops in late-stage female AIDS patients.

In males, Estrogen production can increase due to the AIDS virus, which can inhibit the healthy production of Testosterone. Where Healthy levels of Estrogen are a benefit for female AIDS patients, in Men, it can significantly exacerbate the physical symptoms of AIDS if the body begins to forfeit Testosterone for Estrogen.

The AIDS virus has been highly correlated with clinically low levels of endogenous Testosterone in males. Studies have shown that around 30 percent of AIDs patients suffer from hypogonadism, which is correlated to elevated body fat, cholesterol, and diminished muscle mass in these same patients. Testosterone Replacement Therapy is quickly revealing itself as a hallmark in AIDS therapy.

Central Hypogonadism

Whereas the above forms of hypogonadism are all associated with primary hypogonadism, there are a number of other forms of Testosterone and Estrogen Deficiency that result from another form of deficiency known as Central Hypogonadism.

Central Hypogonadism results from a dysfunction of the pituitary gland or the hypothalamus, which prevents the testes or ovaries from receiving the signal to produce primary sex hormones. Central Hypogonadism is also referred to as Secondary Hypogonadism as well. Causes of Secondary Hypogonadism include:

Tumors:

Prolactinoma

The tumors which most often lead to Hypogonadism are prolactinoma and craniopharyngioma. Prolactinoma is a benign tumor that sometimes develops on the pituitary gland in adult patients. This form of brain tumor can occur in both men and women but is 5 times more likely in female patients. In females, prolactinomas are usually small and do not grow larger over time. When prolactinoma occurs in male patients, the tumors often develop to a much larger size and are then known as macroprolactinomas.

In males, these large tumors often develop late in adulthood. In both men and women, the symptoms of this disorder are similar. In females, the breasts become overly tender and begin to lactate at inappropriate even when one is not nursing. In addition, headaches and infertility can also occur as a result. Prolactinoma may also induce early menopause.

Male patients may develop gynecomastia or the development of breasts. Males may be rendered impotent due to the tumor, and both sexes may struggle with diminished libido due to prolactinoma.

A number of symptoms result from the presence of a tumor within the brain, such as fatigue, vision alterations, and nausea. Hormone Replacement Therapy is a necessary treatment valid after the prolactinoma has been treated. Estrogen can exacerbate symptoms related to an existing prolactinoma, but the tumor can also eventually obstruct proper Estrogen levels produced by the Ovaries.

The exact course of therapy also applies to male prolactinoma patients as well. After prolactinoma has been adequately treated, restoring healthy sex hormone production is vital.

Craniopharyngioma

Craniopharyngioma is another brain tumor that significantly affects the production of primary sex hormones. This type of tumor develops in very close proximity to the pituitary gland. It is uncommon in adults and typically develops in childhood between the ages of five and ten. Although the tumor is benign in form, it can lead to significant symptoms that can disrupt a child's life and his or her family.

The tumor builds pressure in the brain, has the potential to endanger eyesight, and also prohibits the proper function of the pituitary gland. Although craniopharyngioma often affects testosterone and estrogen production in children, it can also adversely affect overall behavior and other mental and biological functions controlled by the pituitary gland.

Tumor Treatment

Surgery, or a mixture of surgery and radiation therapy, is generally the best option to eliminate the benign tumor. Because of the significant hormonal disruption caused by this form of tumor, after the growth has been excised, Total Hormone Replacement Therapy is required for some time after surgery to restore healthy function.

Because of the sensitive age at which this tumor develops, it is essential to initiate HGH Hormone Replacement Therapy as well as Testosterone or Estrogen Replacement Therapy to ensure that proper growth and physical development still occur as the pituitary gland recovers from obstruction.

Hemochromatosis

Hemochromatosis is another cause of pituitary insufficiency. This medical disorder results from excess iron accumulation in the body due to a hereditary defect that causes the body to absorb dangerous levels of iron from what would otherwise be a regular diet.

Particular organs such as the liver, pancreas, and heart serve as reservoirs for excess iron collected through the bloodstream, which can lead to damage if untreated. This iron build-up also occurs in the testicles and the pituitary gland, leading to both primary and secondary forms of hypopituitarism.

In patients with damage to the testes or the pituitary gland, Testosterone Replacement Therapy is an effective means to improve the outcomes for this subset of patients, increasing bone strength and restoring sexual desire, and boosting energy levels.

Radiation and Surgery

As was described earlier, cancer and tumor radiation techniques and surgical procedures can sometimes disrupt the body's ability to produce sufficient levels of sexual hormones.

Whether it be the removal of a pituitary tumor or treating a form of brain cancer located near the pituitary or hypothalamus, these medical techniques can injure or lead to the destruction of brain tissue that controls the secretion of estrogen and testosterone.

Brain trauma and internal hemorrhaging can create pressure on the pituitary or hypothalamus, which prevents them from performing their endocrine regulating functions.

Long-term malnutrition can also induce hypogonadism because, without a proper diet, the body will not receive all the energy and building blocks needed to perform all the functions that keep us healthy and well-maintained.

Kallman Syndrome

Kallmann syndrome is another hereditary genetic disorder that is a form of secondary hypogonadism. Kallmann syndrome is a disorder that affects both young boys and girls, preventing them from starting puberty or, in some cases leading to only partial completion of adult sexual development. The defining non-sexual trait of this disorder is that it wholly or partially leads to the absence of a patient's sense of smell.

Kallmann syndrome primarily affects the hypothalamus and is an excellent example showing how disruptions to the hypothalamic-pituitary-gonadal axis can lead to a cascade of hormone deficiency.

Genetic abnormalities in these patients prevent the hypothalamus from secreting a vital precursor hormone known as GnRH (Gonadotrophin Releasing Hormone), which leads to direct deficiencies of luteinizing hormone and follicle-stimulating hormone.

These deficiencies prevent the young male testes or female ovaries from getting the hormonal message to produce testosterone at the beginning of puberty, which can completely disrupt the normal growth process if not recognized promptly and treated effectively.

Testosterone and Estrogen Hormone Replacement Therapy is a primary treatment for this form of hypogonadism, provided around the time when puberty should begin and allowing these patients to develop normally into adulthood. Hormone Replacement Therapy is required to treat this disorder throughout the lifespan of both sexes to maximize the health outcomes for these patients. It also helps to sustain fertility throughout adulthood.

Hypogonadism Symptoms in Children

The primary symptom of hypogonadism in childhood is an interference with the natural puberty cycle. This means that the breasts will not develop fully in girls, and menstruation will not begin. Body hair will also not develop properly.

In addition, the delay or absence of puberty will also lead to Human Growth Hormone deficiency because although Human Growth Hormone is the primary actor which induces growth. Puberty signals the pituitary to begin pumping out Human Growth Hormone at a peak capacity.

In young boys, testosterone deficiency prevents the body from achieving the many monuments of male puberty. In addition to spurring growth during puberty in both sexes, males do not develop the enhanced muscle mass that separates them from their feminine counterparts. In addition, the voice does not deepen and change, and facial and body hair do not develop properly.

Adult-Onset Hypogonadism

Male Hypogonadism: Low-T, or Testosterone Deficiency

Although Adult-Onset Hypogonadism is not nearly so devastating, there are still significant and life-altering changes that result. In males, hypogonadism results in reduced muscle mass as well as fatigue. Body hair and beard hair are thin or have stopped growing.

In some cases, without the correct levels of Testosterone, the underlying estrogen created by the adrenal glands starts to become more dominant, which can result in the formation of male breasts, clinically known as gynecomastia. Hypogonadism also wreaks havoc on the reproductive system as well.

Patients who suffer from low testosterone have reduced sperm counts as a result. In addition, erectile dysfunction is a significant issue in male patients suffering from hypogonadism.

Male patients suffering from erectile dysfunction are encouraged to get their testosterone levels evaluated by a physician because ED drugs such as Cialis and Viagra only serve to cure the symptoms of this unfortunate disorder. In contrast, complete Testosterone Replacement Therapy can create much more beneficial results in the long term.

Female Hypogonadism Symptoms: Estrogen Deficiency and Low Testosterone Production

Hypogonadism that occurs before menopause is a clinically treatable disorder in female patients. Two symptoms of Estrogen Deficiency are missing periods and a lack of menstruation. In addition to this, many of the hallmark symptoms of menopause begin to appear earlier than they should, including hot flashes and cold sweats.

Hot flashes directly result from the female body craving much-needed estrogen and are one of the primary warning signs of estrogen deficiency. The hair on both the head and the body begins to thin and fall out in many cases due to female hypogonadism.

Estrogen Deficiency is easily treatable with Estrogen Replacement Therapy. It can be used as a therapy to alleviate the symptoms of menopause as the female body adjusts to the changes.

Hypogonadism caused by Tumor Growth

Suppose Testosterone or Estrogen Deficiency results from a tumor in the brain on or adjacent to the hypothalamus or the pituitary. In that case, symptoms such as visual blurring or total vision loss may occur due to pressure on the ocular nerve. In addition, intracranial pressure resulting from the tumor may bring about headaches ranging from minor yet persistent to significant migraines.

Tumors of this kind also result in general disruptions of the hypothalamus and pituitary functions. Testosterone and Estrogen regulation is only one of the many endocrine functions these two brain structures serve, so an outside source of disruption such as a tumor will cause a wide variety of issues in many cases.

Hypogonadism and Anorexia

Individuals who struggle with anorexia also have significant issues with hypogonadism due to their poor diets. Calorie-restricted diets have been scientifically correlated with Low-T and Estrogen deficiency. Anorexia has also been linked with osteoporosis and skeletal degradation, resulting in increased breaks and fractures.

Testosterone and Estrogen are keys to healthy bone maintenance, and when diet restricts the production of sex hormones, the bones suffer significantly. Although most patients who deal with anorexia are female, the effects of anorexia on the endocrine system of both males and females are very significant and, in some cases, can persist for an extended period after anorexia is officially diagnosed and controlled.

How is Hypogonadism Diagnosed?

There are a number of accurate and effective tests which can help provide a proper diagnosis of both Low Testosterone and Estrogen. The primary means by which Low Testosterone is diagnosed is via a test of Total Testosterone. From a single blood sample, it is possible to reveal the body's present Testosterone level. The healthy range for normal testosterone is between 260 and 1080 nanograms per decilitre. You likely quickly realize that this is a wide range. This is because what is considered average declines with age.

A patient in his 40s or 50s likely would experience many symptoms of testosterone deficiency at 260 ng/dl, but a patient in his 80s would experience far fewer symptoms. It is all relative to age. Another Anti-Aging Specialist provides a helpful rule of thumb when considering healthy Testosterone Levels.

At the age of 25, when Testosterone levels are near their peak, a healthy testosterone level should be around 700 ng/dl. For every ten years, you are over the age of 25, and your optimal level of Testosterone will like to be 50 ng/dl lower. At 45, your optimal level will be around 600, still far higher than the minimum normal of 260 ng/dl.

Healthy Testosterone Levels Vary

However, these measures of Healthy and Low Free Testosterone levels are not to be taken as gospel. Patients of the same age can have a significant level of differentiation among their healthy testosterone levels. Low-T is not all about a complex number. Some men are optimized to function properly at lower levels of Testosterone, and the hallmark of Testosterone Deficiency is the outward display of symptoms.

Women and Testosterone

Women need much less Testosterone than men to be healthy, but Testosterone secretion by the adrenal glands is still essential for health and sexual maintenance. In women, a healthy level of Testosterone is somewhere between fifty and seventy ng/dl.

This is significantly lower than what is considered healthy for males, but it is still necessary to maintain optimum hormone function in the female body. In women, Low-T is associated with low libido and hindered sexual function. In addition to sexual disruption, Low-T is also associated with loss of muscle tone, increased instances of depression, fatigue, and insulin resistance.

Healthy Estrogen levels in women vary significantly depending upon the period of their menstrual cycle. Estrogen levels are measured indirectly by taking a blood sample and testing estradiol levels in the bloodstream.

In the initial phase of the cycle, estradiol levels will be around 30 picograms per milliliter. As the follicle which contains the egg begins to develop, estradiol levels rise significantly, and normal ranges anywhere from 150-300 pg/ml.

By the time a woman's period occurs, estradiol levels drop to around fifty to one hundred pg/ml. After menopause occurs, estrogen levels in the blood decline sharply. Normal estradiol levels among postmenopausal women remain flat at around ten mg/dl. Although this level is relatively low, there is still a vital need for the hormone. Women with estradiol levels lower than ten pg/ml should consider Estrogen Replacement Therapy.

Estrogen and the Male Body

Males also have a certain level of Estrogen, which is necessary for proper function. As long as the male body produces appropriate testosterone levels, there is no issue with low estrogen because the male body can convert excess Testosterone into Estrogen and other female sex hormones.

The primary problem with males regarding estrogen is that as men age, they begin to produce more Estrogen than they need, which can lead to emasculating effects and elevated adverse health risks. Normal estrogen for an adult male is similar to that of post-menopausal women.

Healthy males have between ten and fifty pictograms per milliliter of estradiol in the blood. If levels are higher than fifty, a man may need to look into estrogen blockers to prevent their Testosterone from converting too readily into female hormones.

Elevated estrogen levels in males are linked to benign prostate growth (clinically referred to as prostatic hypertrophy), prostate cancer, and gynecomastia.

What is the root cause of Hypogonadism?

If you test positive for Low Testosterone or Estrogen Deficiency, the next step is to determine whether your deficiency results from Primary or Secondary Hypogonadism. One means of testing for Secondary Hypogonadism is by testing serum levels of Luteinizing Hormone.

This hormone is vital in the proper endocrine function of both males and females. In males, Luteinizing Hormone is responsible for stimulating the Leydig cells in the testicles.

Luteinizing Hormone and Hypogonadism

These cells are the primary manufacturing centers for Testosterone in the male body. Luteinizing Hormone acts as the intermediary hormone that sends messages from the Pituitary to the Testes to trigger production. Suppose a male patient suffers from low levels of Luteinizing hormone. In that case, their healthy testicles will not receive the memo initially sent from the hypothalamus to provide the body with much-needed Testosterone.

Although the target destination of Luteinizing Hormone in female patients is different, the general pathway is the same. In this case, Luteinizing Hormone is an intermediary which sends the message to initiate ovulation.

When it is time to begin ovulating, the pituitary gland sends a relative flood of Luteinizing hormone to the ovaries to begin this cycle phase. Suppose a woman does not produce adequate levels of Luteinizing hormone. In that case, she does not meet the threshold required to ovulate, and she will miss her period and suffer from the effects of hormone instability.

FSH and Hypogonadism

Follicle Stimulating Hormone is also an essential precursor to estrogen and Testosterone production. As can be deduced by the name, one of the primary purposes of Follicle Stimulating Hormone is to encourage the production of ovarian follicles, one of which will be used to house the egg cell that will be released from the ovary during menstruation.

Follicle Stimulating Hormone is also necessary to develop both egg and sperm cells, preparing them for their final stage in which they will be viable for reproduction. Low levels of Follicle Stimulating Hormone represent the same sort of Secondary Hypogonad dysfunction as Luteinizing hormones, and often, the two hormones are deficient simultaneously.

GnRH and Hypogonadism

Gonadotropin Releasing Hormone (abbreviated GnRH) is another hormone that directly correlates with the proper function of the testes and ovaries. GnRH is the intermediary hormone that the Hypothalamus uses to direct the Pituitary gland to produce more Estrogen and Testosterone. GnRH travels to the pituitary via a series of blood vessels known as the hypophyseal portals.

A common way that this form of Secondary Hypogonadism is diagnosed is via a simple test. A physician injects GnRH and monitors the patient to observe if their FSH or LH levels elevate to normal. If FSH and LH levels return to normal, then Testosterone Deficiency or Estrogen Deficiency results from a deficiency of GnRH.

What if it is not Hypogonadism?

Blood tests for alternate pathologies that can produce symptoms similar to Hypogonadism are also conducted. Anemia, for example, can lead to many physical complications such as chronic fatigue and weakness that can also be attributed to Hypogonadism. A number of different blood analyses occur, so an accurate diagnosis of Hypogonadism can also occur.

Visual Imaging and Hypogonadism

In some cases, if blood testing produces evidence that there may be some sort of structural malfunction, a sonogram of the testes or ovaries may be conducted to monitor for tumors and abnormal growths. If blood testing suggests hypogonadism results from secondary complications in the hypothalamus or pituitary, a CT scan or MRI can provide a clear view of potential brain tumors.

The key to solving Hypogonadism is to source the root cause of the endocrine malfunction and treat the disorder in a way that allows the body to return to a level of maximum hormonal balance.

Treatment of Hypogonadism

For many forms of hypogonadism, including Testosterone Deficiency and Estrogen Deficiency, simple Hormone Replacement Therapy is the most effective course of action. Estrogen Replacement Therapy is generally provided via pill or patch. On the other hand, testosterone HRT is not effective via pill and is usually provided by injection, patch, or cream.

For women who undergo hysterectomy, a combination of Estrogen HRT and Progesterone are generally prescribed to maintain hormonal balance and prevent endometrial cancer. Testosterone Replacement Therapy is highly effective for hypogonadal women to bolster sexual desire and relieve depression.

Suppose hypogonadism results from an issue such as a brain or prostate tumor preventing proper endocrine function. In that case, a few different options can be used to restore the proper release of the sexual hormones. In the case of small, benign tumors such as prolactinoma, certain medications can inhibit the abnormal stimulation, thus restoring regular hormone release.

In the case of larger or obstructive tumors, surgery is sometimes the best option. Once the tumor has been surgically removed, it no longer adversely pressures or directly affects the pituitary, and the gland can return to normal function.

In other cases, radiation therapy can be used to achieve the same effect. There is always a risk with radiation therapy or surgery that complications can arise that damage the pituitary, which will result in a need for Estrogen or Testosterone Hormone Replacement Therapy to sustain normal function after the surgery, but these techniques are only used with the risk of having the tumor outweigh the risks of having it removed or destroyed.

HGH and Testosterone Hormone Replacement Therapy HRT

For patients who suffer from severe or total hypogonadism, Hormone Replacement Injections or Medications may be required to stimulate the normal functions of the reproductive system, such as sperm production and ovulation.

In conjunction with any form of Hormone Replacement Therapy, steps will be taken to enhance the effectiveness of treatment through nutritional and general health maintenance as well.

Hypogonadism, in most cases, is simple to treat with Hormone Replacement Therapy. For those who suffer from Adult-onset Hypogonadism, with proper Hormone Replacement, life will generally continue with few to no issues that result from therapy.

Symptoms of Hypogonadism in Adults

Symptoms of Female Hypogonadism

In females, Adult-onset hypogonadism can lead to infertility. In addition, it can lead to hot flashes and cold sweats, which are symptoms of Estrogen withdrawal. Mood changes can also result from Estrogen Deficiency, as the hormone plays a significant role in female mood stability. Sexual issues can result from female hypogonadism as well.

Libido can erode or even disappear entirely. Also, the sexual ability can be hampered as well.

Female adult-onset hypogonadism can lead to vaginal dryness and pain during sexual activity. Bone health is also adversely affected as estrogen and testosterone play a role in calcium maintenance in the bones. Osteoporosis is one of the most common symptoms of estrogen deficiency. Low Estrogen levels also increase the risk of heart disease in women.

It should be noted that, unlike men, women undergo severe hypogonadism due to the physiological process of menopause.

Although the symptoms and treatments are similar, menopause is not considered pathological. Adult-onset hypogonadism in females is a diagnosable disorder that can mimic many of the negative aspects of menopause.

Estrogen Replacement Therapy is viral among women who experience menopause early, which is clinically referred to as premature ovarian failure. Estrogen Replacement Therapy after menopause is a valid form of Anti-Aging Therapy that decreases the risk for heart disease and osteoporosis while increasing libido, sexual ability, and overall well-being. It also provides many other benefits as well.

Women with a significant family history or a genetic predisposition toward heart disease and breast cancer should consider the risk of Estrogen Replacement Therapy before starting treatment because there is a slight but significant increase in the chance that these medical issues arise when taking Estrogen HRT.

Symptoms of Male Hypogonadism

In males, a number of symptoms arise from Adult-onset hypogonadism. Although men do not go through a change as significant as menopause as a result of the aging process, Testosterone levels begin to decline at a marked rate after age thirty.

Testosterone Deficiency can lead to loss of libido and infertility. Testosterone production is highly correlated with sperm production, and if the male body does not receive enough Testosterone, it can become harder to conceive.

Testosterone deficiency also leads to erectile dysfunction and impotence because Testosterone is the critical factor in the cascade of physiological reactions which lead to and sustain an erection. As with women, the primary sexual hormone also plays a significant role in bone health and prevents the onset of osteoporosis.

In males, Testosterone also plays a central role in muscle health because Testosterone is the crucial difference between males and females, leading to the formation of enhanced muscle size and strength.

Mood instability and depression can also result from Testosterone Deficiency as well. Extreme Testosterone Deficiency in males is linked to a certain level of feminization which is most frequently characterized by the development of the breast tissue, clinically known as gynecomastia.

Prevention of Hypogonadism

There is no way to prevent Hypogonadism in many cases, such as with tumors. It can only be treated as symptoms arise. Visit your doctor for regular check-ups to catch these disorders as they arise so they can be treated before wreaking significant havoc upon the endocrine system. Initiate Hormone Replacement Therapy as soon as possible after the hormone disruption is alleviated or diagnosed to minimize the effects of Short Term or Permanent Hypogonadism.

Hypogonadism, which results from the natural hormonal decline of aging, can be slowed with healthy and conscientious living. Poor lifestyle choices such as smoking can lead to Premature Aging, which is directly correlated with a hormonal decline. Healthy hormone production is also dependent upon a healthy and nutritious diet. The building blocks of proteins are all contained within the foods we eat and the vitamins and minerals.

Also, obesity and overeating can greatly hinder natural Testosterone levels. Fat cells can convert Testosterone into estrogen and estrogen derivatives, and the more obese a man is, the greater level of Testosterone his body converts into estrogen. In patients receiving Hormone Replacement Therapy with Testosterone or Estrogen, the benefits of therapy are amplified by living a healthy lifestyle.

The Conscious Evolution Institute: Professionals in the Field of Hormone Replacement Therapy

Our staff at the Conscious Evolution Institute is clinically trained and board certified in Hormone Replacement Therapy. Our qualified staff of Physicians and Specialists have years of experience in diagnosing and treating mild to severe Adult-onset Hypogonadism in both men and women.

We provide complete remote diagnostic services and have the capability to provide and deliver prescriptions to patients anywhere in the United States. We have affiliates in your area who can aid us in proper diagnosis.

If you qualify, we can have high-quality Testosterone or Estrogen Replacement Therapy delivered right to your doorstep via overnight FedEx shipping.

In addition to providing treatment for Hypogonadism via Estrogen and Testosterone HRT, we provide many other Hormone Solutions such as HGH and Sermorelin Acetate Therapy. Hormone Replacement Therapy isnâaot for everyone and some patients respond more vigorously than others, but we feel that if you contact us, you will never regret it!

Weâaove changed the lives of thousands of men and women across the country for the better, and we want you to be next! Call or contact the Conscious Evolution Institute today.

 

Word Count: 5691